This, again, is consistent with the tumor suppressor role attributed to mir-128-3p [37], which we confirmed by identifying a drop in its expression compared to the level quantified in non-tumor brain tissue, and with the work of Zhang et al., 2019, who reported that a low miR-100 expression correlated with worse clinicopathological characteristics such as Karnofsky Performance Scale and IDH1/2 mutation status [48]. The gene discussed is IDH1; the disease is neoplasm.