Besides its structural properties, Fscn1 was also shown to actively translocate from the cytoplasm into the nucleus of tumor cells and to positively regulate expression of pro-tumorigenic genes, such as the amino-acid transporter solute carrier family 3 member 2 [17] and to promote pro-tumorigenic canonical wingless signaling via activation of activation of focal adhesion kinase [18]. The gene discussed is FSCN1; the disease is neoplasm.