The altered expression of Ribosomal proteins has been linked to multiple LC-related processes, e.g., the downregulation of 60S ribosomal protein L3 (uL3) has been linked to drug resistance in LC cells, and 40S ribosomal protein S6 (eS6) was found to be overexpressed in NSCLC [66,67]. This evidence concerns the gene RPL3 and laryngotracheoesophageal cleft.