On this scenery, it may be feasible that activity in GalR2-NPYY1R heteroreceptor complexes, which, as indicated above, are elevated upon agonist coactivation of GalR2 and NPYY1R [260], may allow for an increased inhibitory tone upon CeA neurons leading to a reduction in the anxiogenic CeA output with the consequent diminution of anxiety. This evidence concerns the gene GALR2 and Anxiety.