Based on etiologies in decompensated cirrhosis patients, i.e., alcohol (ALC) and other etiology (NASH and cryptogenic), we have found no difference in the percentage of neutrophils CD11b+CD66b+, CXCR1+, and CXCR2+ on CD11b+CD66b+ cells in decompensated cirrhosis patients w/o and with sepsis (Supplementary Figure S2A). This evidence concerns the gene CEACAM8 and metabolic dysfunction-associated steatohepatitis.