postulated that in preeclampsia, increased levels of pro-inflammatory chemokines in the blood and placenta might bind to the syncytiotrophoblast membrane’s ACKR2 decoy receptor, resulting in an increased concentration of the scavenger on the syncytiotrophoblast cell membranes as a result of its mobilization from the intracellular pool. The gene discussed is ACKR2; the disease is preeclampsia.