CXCR4 and Miyoshi myopathy: Thus, quantification of [68Ga]PentixaFor accumulation may then allow to estimate the efficacy of non-radioactive CXCR4 inhibitory treatments (e.g., with anti-human CXCR4 IgG monoclonal antibodies for MM patients) [65] or to identify patients that would be eligible for treatment with hot CXCR4-directed theranostic radiotracers, such as [177Lu]/[90Y]PentixaTher [46].