KLRK1 and chronic myelogenous leukemia, BCR-ABL1 positive: In summary, CML-RAE-1γ-Dex contributed to immune function in a NKG2D/NKG2D-L (RAE-1γ)-dependent manner, as blocking NKG2D signaling with a neutralizing antibody against NKG2D reduced the cytotoxicity of NK cells and T lymphocytes.