To determine whether the neuroinflammation which occurs in both AD129–133 and PD77,134–137 could produce a similarly increased percentage of DAT+/TH+ PBMCs that could be generalized to neurodegeneration, we repeated these experiments in a cohort of Alzheimer’s diseases (AD) patients with no movement disorder, psychoses, and no use of dopaminergic drugs (i.e. neuroleptics, antidepressants). Here, SLC6A3 is linked to Alzheimer disease.