The most common forms of amyloidosis infiltrating the human heart (cardiac amyloidosis, CA) are (a) immunoglobulin light chain (AL) [1] and (b) transthyretin amyloidosis (ATTR) [2] which in turn comprises two subtypes: a hereditary form (ATTRv) caused by the presence of a TTR gene mutation and a wild-type form (ATTRwt) caused by age-related instability of wild-type TTR [3]. This evidence concerns the gene TTR and cardiac amyloidosis.