Anderson and Potter demonstrated the molecular association of plasma cell neoplasms and ASIA-MO in BALB/c susceptible murine models with pristane intraperitoneal induction as an enhancer of the innate immune system with arthritis, lupus-related autoantibodies, and IgA-secreting plasma cell neoplasms as a result [21, 22]. The gene discussed is CD79A; the disease is arthritic joint disease.