Because SLC7A11 is a recognized target in ferroptosis, blocking it will directly rein in the uptake of Cys, which can be utilized for GSH synthesis in the antioxidant process (28). Moreover, SLC7A11 has been found to be highly expressed in non-small cell lung cancer (NSCLC) with a poor prognosis (29). Here, SLC7A11 is linked to non-small cell lung carcinoma.