To this regard, the fatty acid transport protein 2 (FATP2) was recently identified as a regulator of the suppressive capacities of G-MDSCs; it is a neutrophil membrane protein implicated in the trafficking of lipids and it is overexpressed by G-MDSCs in tumor-bearing mice compared to “classical” neutrophil isolated in healthy mice. The gene discussed is SLC27A2; the disease is neoplasm.