In COVID-19 patients with a higher level of IL-6 and IL-10; increased CRP; elevated levels of ALT, AST, and creatine kinase; increased neutrophils; and decreased lymphocytes, the clinical coarse of disease is more severe and the mortality rate is considerably higher (Huang C. et al., 2020; Wu and Yang, 2020).In contrast, a high level of IL1B, IFNγ, IP10, and MCP1 probably leads to the activation of the T-helper-1 (Th1) cell responses and increases the secretion of T-helper-2 (Th2) cytokines (IL4 and IL10) that suppress inflammation (Mescher et al., 2006). The gene discussed is CCL2; the disease is COVID-19.