The results revealed that the necroptosis-associated lncRNA prognostic model modulated immune-associated disease and processes such as antigen processing and presentation, homologous recombination, allograft rejection, graft versus host disease, primary immunodeficiency, NK cell-mediated cytotoxicity, cytokine receptor interaction, intestine-based immune-networking for IgA generation, and systemic lupus erythematosus (Figure 7). This evidence concerns the gene CD79A and graft versus host disease.