MAGEC1 and neoplasm: Our findings that the CTAG1B/A network is enriched with transcription factors (Figures 3C, 5) and its ability to compensate for the absence of the yeast transcription factor, PCC1P, support our hypothesis that CTAG1A complexes (likely with MAGEC1) are part of gametogenic germ cell transcription programs that could also be recruited to support tumor growth.