OAS1, OAS2, OAS3, IL10RB, FEM1A, GART, SON, and IFNAR2 were significantly (FDR < 0.05) upregulated in lymphocytes and monocytes from COVID-19 patients compared to healthy controls, while TNFAIP8L1 was significantly downregulated in monocytes from COVID-19 patients (Fig. 3A, B). This evidence concerns the gene OAS1 and COVID-19.