In addition, the expression of tRFLys-CTT-010 is increased in TNBC and interacts with the glucose-6-phosphatase catalytic (G6PC) subunit to regulate cellular lactate production and glycogen consumption levels, which promotes the proliferation of cancer cells, suggesting that tRFLys-CTT-010/G6PC and the glucose metabolism regulatory axis may be a new therapeutic target for TNBC [71]. Here, G6PC1 is linked to cancer.