While comparing CCL2 protein levels in hypoxia treated miR‐210‐3p inhibitor transfected cells (Fig. 2M) with the cells incubated with 5‐Azacytidine in the presence of hypoxia (Fig. S3L), we noticed that hypoxia‐induced CCL2 expression primarily governs through the HIF‐1A–miR‐210‐3p axis rather than the HIF‐1A–DNMT axis in A549 lung cancer cells. This evidence concerns the gene CCL2 and lung cancer.