For example, Vlaskamp et al. [27] in a cohort of 57 SYNGAP1 patients found that 98% had epilepsy which were characterised as eyelid myoclonia with absences (65%), myoclonic seizures (34%), atypical (20%), and typical (18%) absences and atonic seizures (14%), which were triggered by eating in 25%. The gene discussed is SYNGAP1; the disease is epilepsy.