al. emphasized the involvement of SNAI2 in glioma cell stem-like properties; A decrease in SNAI2 elicited by overexpressed miR-124 contributed to a reduction in the CD133(+) cell subpopulation and decreased Nestin expression, while suppressing the in vivo tumorigenicity and invasion of glioma cells [26]. This evidence concerns the gene PROM1 and glioma.