Consistent with our previous findings, cardiac BCAA levels were significantly higher in the post-infarction hearts of the mice fed BCAA compared to the vehicle group (supplementary Fig. 2b).6,7 Intriguingly, dietary BCAA intervention significantly reduced the retention of ADSCs in the post-infarction heart, as observed in the PP2Cm KO models (supplementary Fig. 2c). The gene discussed is PPM1K; the disease is infarction.