Similarly, analysis of the Taylor et al. Memorial Sloan-Kettering Cancer Center (MSKCC) 2010 data set (see Supplemental Methods) demonstrated that transcript levels of Hsp60, Hsp10, and ClpP (Figure 4, F–H) were higher in PCa compared with the MN tissues, whereas LONP1 expression did not change (Supplemental Figure 6E) and PARL expression was reduced (Supplemental Figure 6F). The gene discussed is PARL; the disease is posterior cortical atrophy.