MTOR and congenital primary aphakia: In our phosphoproteomics study on mouse brains, we found that U50,488H activated the mTOR pathway in the striatum and cortex, but nalfurafine did not, and inhibition of mTOR with rapamycin blocked U50,488H- and MOM-SalB-induced CPA, indicating that the mTOR pathway is involved in U50,488H- and MOM-SalB-induced CPA (Liu et al., 2019).