Other alarmins that can trigger type 2 inflammation, such as IL-25 and IL-33, as well as cytokines involved in inflammation and lung tissue repair such as IL-22, TNF, and TGF-β, have not been extensively addressed for their participation in morbidity and sequela of COVID-19 (Johnson and others 2013; Roan and others 2019; Wu and others 2019). This evidence concerns the gene TGFB1 and COVID-19.