CXCR3 and neoplasm: CXCL9, CXCL10, CXCL11, and CXCR3 recruited and activated immune cells, such as CD8 + T-cells, to suppress tumor progression through CXCL9,-10−11/CXCR3 axis, and they were down-regulated in the high-GATA3 group (27).