This study also preliminarily explored the potential molecular mechanism of BSG in various human cancers and discussed the sensitivity of some drugs to BSG. In the GSEA, BSG was closely related to olfactory transduction, xenobiotics metabolism by cytochrome P450, hematopoietic cell lineage, drug cytochrome metabolism P450, JAK-STAT signaling pathways, Leishmania infection, and pentose and glucuronate interconversions. This evidence concerns the gene SOAT1 and cancer.