VIM and cancer: Paclitaxel (47), Artemisinin (39), Dihydroartemisinin, and Apigenin (66), could induce Wnt, Notch, and P38 signaling pathways, upregulate the expression of SH2-containing protein tyrosine phosphatase 1 (SHP-1) (67), RECK, β-Catenin, Vimentin, Claudin-1, ZO-1, downregulate the level of E-cadherin, p-STAT3, mucin1, Gli2, Vimentin, Snail, and Twist, therefore, inhibiting EMT to suppress cancer cell proliferation, migration and invasion.