ACE2 and COVID-19: Heightened expression of ACE2 in senescent endothelial cells indicates there is a pathway to increased viral infection that contributes to endothelial injury, inflammation, and dysfunction in COVID‐19 (Bonaventura et al., 2021; Conde et al., 2020; Huertas et al., 2020; Jin et al., 2020; Varga et al., 2020).