This modelling (Fig. 2C) showed no statistically significant changes in the mobile fraction or half-time of recovery of GFPMBNL1 in SGs in DM1 cells compared to controls, suggesting that SGs, once formed, are not defective in DM1 cells in terms of MBNL1 dynamics. This evidence concerns the gene MBNL1 and myotonic dystrophy type 1.