In line with previous findings that DGCs cooperate with GSCs to promote tumor growth (Wang et al., 2018), we then evaluated the ability of DGCs silenced for FMOD to support the growth of tumors initiated by GSCs in co-implantation experiments in a syngeneic mouse model using GSCs and DGCs derived from DBT-Luc glioma cells. The gene discussed is FMOD; the disease is glioma.