CXCL10 and infection: An assessment of the expression of a panel of chemokines in lung tissue homogenates of mice on days 2 and 5 post infection revealed that while both S. aureus and HSV-1 infection triggered the expression of CCL4, CXCL9, and CXCL10, by day 5 p.i., only CXCL9, which elicits its chemotactic function by interacting with CXCR3, was still elevated in the lung tissue in both infection models (Fig. 4d, e).