An assessment of the expression of a panel of chemokines in lung tissue homogenates of mice on days 2 and 5 post infection revealed that while both S. aureus and HSV-1 infection triggered the expression of CCL4, CXCL9, and CXCL10, by day 5 p.i., only CXCL9, which elicits its chemotactic function by interacting with CXCR3, was still elevated in the lung tissue in both infection models (Fig. 4d, e). This evidence concerns the gene CXCR3 and infection.