ITGAM and malaria: Taken together, these results suggest that late IL‐6 induces and expands CD11b+Ly6Chi proinflammatory monocytes, which in turn mediate the inhibition of adaptive immune responses against lethal malaria N67C infections; the depletion of CD11b+Ly6Chi proinflammatory monocytes restores the anti‐malaria immunity and prolongs mouse survival.