IFNA1 and parasitic infectious disease: However, N67C‐infected Il6–/– mice treated with IFN‐α/β at 18 h p.i. had markedly reduced parasitemia levels and increased survival rates compared with N67C‐infected WT mice (Figure 3C,D), suggesting that both an early IFN‐α/β treatment and the blockage of late IL‐6 production are necessary and sufficient to generate strong immunity against N67C infections.