Recently, severe SARS‐CoV‐2 infection has been linked to a unique milieu of LMs with the loss of immune‐regulatory PGs and RvE3 and the increased products of ALOX5 and CYP24 while another study highlighted rise of proinflammatory eicosanoids and docosanoids in the lungs of intubated COVID19 patients.25 This evidence concerns the gene CYP24A1 and COVID-19.