Studies on LPL gene polymorphisms show variants that reduce LPL serum activity, increasing hypertriglyceridemia and decreasing HDL levels, with a probable accumulation of chylomicrons and VLDL at the arterial level leading to a greater predisposition to preeclampsia [72] or even related to more severe disease presentations such as HELLP (hemolysis, elevated liver function and low platelets) [73]. This evidence concerns the gene LPL and hypertriglyceridemia.