Although glucose transporter 2 (GLUT2) is considered the main sugar transporter relevant to liver function [123], Fukuzawa et al. [124] have reported exacerbated hepatic steatosis induced by diminished sodium-dependent fructose uptake in SGLT5-deficient mice, suggesting the potential use of this gene as an indirect biomarker of liver health. This evidence concerns the gene SLC2A2 and Hepatic steatosis.