This evidence suggested that, in our radioresistant (RR) cervical cancer cells (CaSki and C33A), suppressed miR29a might result in a loss of the inhibition of these predicted anti-apoptosis genes (BCL-2, BCL2L2, MCL-1, and AKT1) or inhibition of pro-migration genes (CD44, MMP-7,-16, and -20), and thus RR-cell clones tended to exhibit a decreased apoptosis rate or enhanced cell-migrative properties.Taken together, of interest, the most well-documented function of microRNA-29a (miR-29a) from the literature is its role in the prevention of tissue fibrosis [38,39,40]. Here, AKT1 is linked to cervical cancer.