Studies of placental glucose transporters’ expression in human pregnancy affected by IUGR have had conflicting results; placenta GLUT1 and GLUT3 gene expression has been reported to be not different between IUGR and non-IUGR fetuses or trophoblasts isolated from term placenta that demonstrated increased GLUT1 and GLUT3 mRNA [14]. This evidence concerns the gene SLC2A1 and fetal growth restriction.