APOA1 and glomerulonephritis: Our findings of elevated urinary proteins, which could result from tubulointerstitial inflammation/injury (KIM-1 [56], ApoA1 [47,57], EGF-R [58], AGT [59]), further support that IgAVN includes some degree of tubular inflammation in addition to the glomerulonephritis as previously proposed by Williams et al. [12].