ACTA1 and idiopathic pulmonary fibrosis: Immunohistochemical studies confirmed strong induction and predominant localisation of HDAC1, -2, -3 and -8 in α-SMA expressing myofibroblasts of fibroblast foci, but also revealed robust expression of all Class I HDACs in abnormal, hyperplastic bronchiolar basal cells at sites of aberrant re-epithelialisation in IPF.