The HR repair mechanism (HRR) involves multiple proteins, and germline BRCA1/2 mutations are the most well-known genetic component of HRD in ovarian cancer, accounting for 10–15% of cases, especially for high-grade serous carcinoma (HGSOC), in which they account for 20–30% of patients [17,18,19]. Here, BRCA1 is linked to ovarian carcinoma.