CXCR2 and acute myeloid leukemia: When we continued the culture of GILT-treated MV4-11 cells by medium change and replating for another week, we found that there was a continuous increase in CXCR2 proteins at both the surface and intracellular locations of survived and recovered blasts (flow cytometry histogram, Supplementary Figure S1B), suggesting its potential role in the relapse of FLT3mut AML.