In the first step, we analyzed cDNA microarray datasets connected with acute allergic airway inflammation and revealed top-10 core genes (Ccl6, Ccl9, Ccl12, Timp1, Muc5ac, F5, Cyp2e1, Adra2a, Ear1, Tk1) characterized by (a) nodal disposition within asthma-related gene networks, (b) association with the well-known processes involved in the regulation of asthma, and (c) close connection both with “asthma” and “pulmonary fibrosis” terms in the scientific texts (Figure 2). Here, TIMP1 is linked to pulmonary fibrosis.