Our results indicated that, similarly to the observed genetic obesity experimental model (Zucker rats) (Figure 2B), in the nutritional obesity model, an increase in ALP, bilirubin, vWF, and hyaluronic acid levels was evidenced in both steatotic and non-steatotic grafts of the BD+LT group when compared with the results of the LT group (Supplementary Figure S2). The gene discussed is VWF; the disease is obesity disorder.