The inflammatory response of the organisms involves the proliferation and infiltration of proinflammatory cytokines such as TNF and IL-23/IL-17 in the pathogenesis of psoriasis, while immune cells such as thymic stromal lymphopoietin (TSLP), IL-33, IL-1β and IL-8 characterize the pathogenesis of contact/atopic dermatitis [98]. Here, TSLP is linked to psoriasis.