HDAC6 and retinopathy of prematurity: Apoptosis signal‐regulating kinase 1 (ASK1) has been demonstrated to phosphorylate HDAC6 in response to oxygen changes, blocking the ubiquitination and subsequent proteasomal degradation of HDAC6, thus stabilizing this protein.[7] Since our work showed the critical involvement of the HDAC6‐cilium axis in the pathogenesis of ROP, we next explored whether targeting ASK1 could ameliorate the pathological changes associated with ROP.