The results suggested that the high‐risk group is positively linked with oncogenic pathways related to gliomas that have been proved before, such as angiogenesis,5 E2F target,25 epithelial‐mesenchymal transition (EMT),26 G2/M checkpoint27 and tumour necrosis factor‐transcription factor nuclear factor kappa B (TNF‐NFκB) signalling.28 This evidence concerns the gene TNF and glioma.