IL33 and asthma: While this study differs from the present study in several ways (it utilizes a different, more short term asthma model, only female mice, and murine exogenous leptin), they also measured airway physiology using the FlexiVent system and found that in wildtype mice, exogenous leptin, when combined with IL-33, increased Rrs (equivalent to Rtot), and did not affect Ers, Rn, and H, which is consistent with our findings [24].