Gene set enrichment analysis (GSEA) showed that compared with the SLC24A2-low group, the DEGs in the SLC24A2-high group were mainly enriched in some biological processes including anatomical structure formation involved in morphogenesis, animal organ morphogenesis, antimicrobial humoral response, and biological adhesion (Figure 3(a)) and several KEGG pathways, such as bladder cancer, pathways in cancer, prostate cancer, small-cell lung cancer, and type II diabetes mellitus (Figure 3(c)). This evidence concerns the gene SLC24A2 and urinary bladder carcinoma.