Since the biomarkers included in this study were extracted from studies on ME/CFS and CF-like symptoms in comorbid disorders with duration > 6 months, we may conclude that after resolution of acute inflammation, CAFS-D symptoms are maintained by continued aberrations in the redox, NF-κB, and Wnt axis, increased IL-10 production, and increasing oxidative damage, including secondary autoimmune responses and nitrosylation. Here, IL10 is linked to cystic fibrosis.